[33] Sakaguchi S. 2005. Naturally arising Foxp3-expressing CD25+CD4+ regulatory T cells in immunological tolerance to self and nonself. Nat Immunol.;6(4):345-52.

 

[34] Solomou EE, Rezvani K, Mielke S, Malide D, Keyvanfar K, Visconte V, 2007. Deficient CD4+ CD25+ FOXP3+ T regulatory cells in acquired aplastic anemia. Blood.;

110(5):1603-6.

 

[35] Feng X, Kajigaya S, Solomou EE, Keyvanfar K, Xu X, Raghavachari N, 2008. Rabbit ATG but not horse ATG promotes expansion of functional CD4+CD25highFOXP3+ regulatory T cells in vitro. Blood. ;111(7): 3675-83.

[36] Kojima S, Hibi S, Kosaka Y, Yamamoto M, Tsuchida M, Mugishima H, 2000. Immunosuppressive therapy using antithymocyte globulin, cyclosporine, and danazol with or without human granulocyte colonystimulating factor in children with acquired aplastic anemia. Blood.;96(6):2049-54.

[37] Fuhrer M, Rampf U, Baumann I, Faldum A, Niemeyer C, Janka-Schaub G, 2005.Immunosuppressive therapy for aplastic anemia in children: a more severe disease predicts better survival. Blood. ;106(6) : 2102-4.

[38] Di Bona E, Rodeghiero F, Bruno B, Gabbas A, Foa P, Locasciulli A, et al. Rabbit antithymocyte globulin (r-ATG) plus cyclosporine and granulocyte colony stimulating factor is an effective treatment for aplastic anaemia patients unresponsive to a first course of intensive immunosuppressive therapy.


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